By Novartis Foundation symposium on Anaphylaxis
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J Exp Med 175:289^ 292 Thienes CP, De Monte L, Monticelli S, Busslinger M, Gould HJ, Vercelli D 1997 The transcription factor B cell-speci¢c Activator Protein (BSAP) enhances both IL-4- and CD40-mediated activation of the human e germline promoter. J Immunol 158: 5874^5882 van der Zee JS, van Swieten P, Aalberse RC 1986 Serologic aspects of IgG4 antibodies. II. IgG4 antibodies form small, nonprecipitating immune complexes due to functional monovalency. J Immunol 137:3566^3571 Vercelli D 2001 IgE and its regulators.
This has led to the deaths of patients. On the warning bracelet we write ‘anaphylaxis’. Everyone knows to be frightened of that. But for scienti¢c papers it is a di¡erent ball game. Simons: I would like to comment on the issue of diagnosis. Johannes Ring mentioned that diagnosis of anaphylaxis is easy. However, I’d like to suggest that there is at least one group of patients in whom this isn’t the case, namely infants and pre-school children. I was concerned for many years by the fact that few infants and young children were included in the retrospective studies of anaphylaxis episodes from all triggers in all ages that have been published (Yocum et al 1999, Kemp et al 1995).
More recently we have shown that mast cell chymase released from immunologically activated cardiac mast cells can e⁄ciently convert angiotensin I to angiotensin II. (Marone et al 1998). It is possible that certain mediators such as chymase released from mast cells can play an important role in the homeostatic control of anaphylaxis. Schwartz: It is a fascinating observation that chymase could be a source for generating angiotensin II in tissues. It has been di⁄cult for people doing research on this area to show that such an event is pathophysiologically important.
Anaphylaxis by Novartis Foundation symposium on Anaphylaxis